Rapafusyn closes over-subscribed $44M Series A RapaGlue platform: 8+ billion non-degrading molecular glues Lead program RAP-0001 targets acute kidney injury Spun out of Johns Hopkins - Jun Liu lab Pipeline spans oncology, immunology, renal disease & pain Sean X. Hu, PhD MBA - President & CEO Rapafusyn closes over-subscribed $44M Series A RapaGlue platform: 8+ billion non-degrading molecular glues Lead program RAP-0001 targets acute kidney injury Spun out of Johns Hopkins - Jun Liu lab Pipeline spans oncology, immunology, renal disease & pain Sean X. Hu, PhD MBA - President & CEO
Baltimore, Maryland  /  Drug Discovery  /  Est. 2015

Rapafusyn Pharmaceuticals

A Johns Hopkins spinout building non-degrading macrocyclic molecular glues - and using them to reach the targets other drugs can't.

Rapafusyn Pharmaceuticals, on the Johns Hopkins School of Medicine campus in Baltimore - where a class of natural-product-like macrocycles first discovered in an academic lab is being engineered into medicines.

Series A - $44M Molecular Glues RapaGlue Platform ~21 Employees SIC 2834
$44M
Series A Raised
8B+
DEL Compounds
4
Therapeutic Areas
2015
Founded
The Company

Gluing proteins together, not tearing them apart

Rapafusyn Pharmaceuticals is a clinical-stage drug discovery company based in Baltimore, Maryland. It designs a specific kind of medicine - the molecular glue - and it has taken a deliberately different route than most of the field.

A molecular glue is a small molecule that brings two proteins into contact that would not otherwise meet. In recent years, the best-known glues have been degraders: they recruit a target protein to the cell's disposal machinery and mark it for destruction. Rapafusyn's glues are non-degrading. Rather than sending a target to the shredder, they hold it in place, forming a stable three-part complex that changes what the protein can do.

That distinction matters because it opens a different set of targets. Many disease-driving proteins are considered "undruggable" - they have no obvious pocket for a conventional small molecule to grab, and they sit inside the cell or span its membrane, out of reach of antibodies. Rapafusyn's macrocycles are built to be cell-permeable, which lets them work where those hard targets actually live: protein-protein interfaces, transcription factors, transporters, ion channels, and enzymes.

The company was founded in 2015 on discoveries from the laboratory of Dr. Jun O. Liu, a professor of pharmacology and molecular sciences at Johns Hopkins University and the director of the Johns Hopkins Drug Library. Rapafusyn licensed its foundational chemistry from the university and remains on the School of Medicine campus, drawing on that scientific and clinical ecosystem.

"Rapafusyn and the RapaGlue platform are rewriting what's possible in drug discovery with a new class of macrocyclic molecules." - Dr. Karen Liu, 3E Bioventures
The Platform

Inside RapaGlue

RapaGlue is the engine underneath everything Rapafusyn does - a discovery system for finding, ranking, and refining non-degrading molecular glues at scale.

At its core sits a proprietary DNA-encoded library (DEL) of more than eight billion compounds. Each molecule carries a short DNA "barcode," so the entire collection can be screened against a target in a single pooled experiment and the winners read out by sequencing. On top of that chemistry, the company layers machine learning to guide target selection and to optimize hits for the properties that decide whether a molecule ever becomes a drug: selectivity, potency, cell permeability, and how long it stays bound.

The result is a platform meant to feed a pipeline rather than a single product - the same approach that turned Rapafusyn's $44 million Series A into a bet on a method, not just a molecule.

Scale

8B+ Compounds

A DNA-encoded library large enough to interrogate difficult targets against billions of chemical possibilities at once.

Intelligence

AI-Enabled

Machine learning steers target selection and hit optimization, narrowing an enormous search space to viable leads.

Chemistry

Cell-Permeable Macrocycles

Natural-product-like rings engineered to cross the cell membrane and reach intracellular and transmembrane targets.

Mechanism

Non-Degrading Glues

Modulates protein function by holding targets in a ternary complex - no protein destruction required.

FKBP12
Chaperone
+
RapaGlue
Molecular Glue
+
Target
e.g. ENT1
Ternary Complex
Function Modulated

How a non-degrading glue works: RAP-0001 binds the intracellular chaperone FKBP12 and ENT1, locking them into a single three-part complex.

The Problem & The Pipeline

Starting where the need is highest

Rapafusyn's lead program picks an unglamorous but pointed problem: acute kidney injury in patients undergoing cardiac surgery, a common complication with no approved preventive drug.

That program, RAP-0001, is a selective inhibitor of ENT1 - a nucleoside transporter. As a molecular glue, it binds the intracellular chaperone FKBP12 together with ENT1 to form a ternary complex. In preclinical models it has shown activity against ischemia-reperfusion injury, a major driver of acute kidney injury, and the company presented in vivo data at the American Society for Nephrology's Kidney Week in 2024. RAP-0001 is advancing toward IND-enabling studies.

Beyond the kidney, the RapaGlue platform is expected to feed programs across oncology, immunology and inflammation, and pain - each drawing on the same core ability to reach targets that conventional chemistry struggles to touch.

RAP-0001 - ENT1 inhibitorRenal / Acute Kidney Injury
DiscoveryLead OptIND-EnablingClinic
Oncology programsOncology
DiscoveryLead OptIND-EnablingClinic
Immunology / InflammationImmunology
DiscoveryLead OptIND-EnablingClinic
Pain programsPain
DiscoveryLead OptIND-EnablingClinic

Pipeline stages shown are approximate, based on public statements; RAP-0001 is the most advanced program.

Where It Fits

A different lane in a crowded field

Molecular glues and targeted protein modulation have become one of the most active areas in drug discovery, with companies like Monte Rosa, Kymera, Nurix, Neomorph, and Arvinas building around protein degradation.

Rapafusyn's distinction is its non-degrading approach. Where degraders remove a protein entirely, Rapafusyn's glues change what a protein does while leaving it intact - a mechanism suited to targets you may want to inhibit or redirect rather than eliminate. Combined with macrocycle chemistry tuned for cell permeability and a DNA-encoded library measured in billions, it stakes out a lane that is adjacent to, but distinct from, the degrader crowd.

Commercially, Rapafusyn operates as a B2B biopharmaceutical company: it advances its own wholly owned programs while positioning the platform for partnerships, licensing, and co-development with larger pharmaceutical companies as candidates mature.

"With the successful closing of our Series A financing, we are positioned to propel the RapaGlue platform and to accelerate our mission to deliver transformative therapies to patients in need." - Dr. Sean X. Hu, President & CEO
The People

Who's building it

A small, science-led team pairing Johns Hopkins chemistry and biology with seasoned biopharma leadership.

Sean X. Hu, PhD, MBA
President & CEO

Life-science industry veteran with 25+ years across strategy, drug and diagnostics development, and asset valuation. Previously built and ran clinical-stage biopharma Avotres.

Jun O. Liu, PhD
Scientific Founder

Professor of Pharmacology & Molecular Sciences at Johns Hopkins; co-leads the Kimmel Cancer Center's chemical biology program and directs the Johns Hopkins Drug Library.

Sam Hong, PhD
Head of Platform Sciences

Leads the RapaGlue discovery platform.

Matthew Olson, PhD
VP, Biological Sciences

Oversees the company's biology programs.

Rick Ewing, PhD
VP, Chemistry

Leads macrocycle and medicinal chemistry.

Alex Rabby
SVP, Business Development

Leads partnerships and corporate development.

The Story So Far

A timeline

2015

Founded in Baltimore

Rapafusyn is established, licensing macrocycle molecular-glue technology from Dr. Jun O. Liu's laboratory at Johns Hopkins University.

2024

ENT1 data at ASN Kidney Week

The company presents in vivo data on its ENT1 inhibitor for acute kidney injury at the American Society for Nephrology's 2024 meeting.

2025

Sean X. Hu appointed CEO

A 25-year life-science industry veteran and former Avotres CEO takes the helm as President and CEO.

2025

Over-subscribed $44M Series A

Rapafusyn closes a $44 million round - up from an initial $28M close - to advance RapaGlue and push RAP-0001 toward IND-enabling studies.

Series A - The Cap Table

  • Total raised: $44 million (over-subscribed; includes $24M committed previously)
  • Announced: September 4, 2025
  • New investors: BioTrack Capital, Yonjin Capital
  • Existing investors: 3E Bioventures Capital, Proxima Ventures, Lapam Capital
Worth Knowing

Details that stick

Field Notes

  • Rapamycin roots. The name nods to rapamycin-inspired macrocycle chemistry - glues in the same conceptual family as the FKBP12-binding natural product.
  • Non-degrading by design. Unlike the protein-degradation glues that dominate headlines, Rapafusyn's hold targets in place rather than destroying them.
  • More compounds than people. Its DNA-encoded library holds over 8 billion molecules - more chemical possibilities than there are people on Earth.
  • Same lab, famous library. The science traces to the Johns Hopkins lab that maintains the Johns Hopkins Drug Library, widely used in drug-repurposing research.
FAQ

Questions, answered

What does Rapafusyn Pharmaceuticals do?
It is a clinical-stage drug discovery company that designs non-degrading, cell-permeable macrocyclic molecular glues to modulate hard-to-drug intracellular and transmembrane protein targets through its RapaGlue platform.
What is a molecular glue, and how is Rapafusyn's different?
Molecular glues are small molecules that bring two proteins together. Many glues in development trigger protein degradation; Rapafusyn's are non-degrading, meaning they modulate protein function by holding targets in a ternary complex rather than destroying them.
What is RAP-0001?
RAP-0001 is Rapafusyn's lead program, a selective ENT1 inhibitor that binds FKBP12 and ENT1 to form a ternary complex. It is advancing toward IND-enabling studies for preventing acute kidney injury in cardiac surgery patients.
How much funding has Rapafusyn raised?
Rapafusyn closed an over-subscribed $44 million Series A financing in September 2025, backed by BioTrack Capital, Yonjin Capital, 3E Bioventures Capital, Proxima Ventures, and Lapam Capital.
Where is Rapafusyn located and who leads it?
Rapafusyn is based on the Johns Hopkins School of Medicine campus in Baltimore, Maryland. Dr. Sean X. Hu serves as President and CEO, and Dr. Jun O. Liu of Johns Hopkins is the scientific founder.
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